Table 1.

Baseline characteristics of study population

CharacteristicOverall, N = 55Intense, n = 36VEN/HMA, n = 19P value
Sex, n (%)    .6 
Male 27 (49) 19 (53) 8 (42)  
Female 28 (51) 17 (47) 11 (58)  
Race, n (%)    .8 
White 40 (73) 26 (72) 14 (74)  
Black or African American 10 (18) 6 (17) 4 (21)  
Asian/Pacific Islander 5 (9.1) 4 (11) 1 (5.3)  
Age at diagnosis, mean (SD) 67.3 (4.3) 66.1 (3.9) 69.6 (4.2) .005 
ECOG, n (%)    .29 
0-1 43 (81) 31 (86) 14 (74)  
2-3 10 (19) 5 (14) 5 (26)  
CCI, mean (SD) 5.1 (1.8) 4.5 (0.9) 6.2 (2.6) .003 
Treatment era, n (%)     .001 
Treatment era 1 36 (65) 29 (81) 7 (37)  
Treatment era 2 19 (35) 7 (19) 12 (63)  
WBC count at diagnosis, ×103/μL, mean (SD) 65 (78) 74 (82) 47 (67) .12 
Extramedullary disease present, n (%) 18 (33) 12 (33) 6 (32) .9 
Therapy-related, n (%) 7 (13) 4 (11) 3 (16) .7 
Myelodysplastic-related, n (%) 14 (25) 10 (28) 4 (21) .7 
MDS-associated mutation, n (%) 17 (45) 8 (38) 9 (53) .4 
FLT3-TKD mutated, n (%) 19 (35) 12 (33) 7 (37) .8 
DNMT3A mutated, n (%) 16 (29) 8 (22) 8 (42) .12 
IDH2 mutated, n (%) 15 (27) 7 (19) 8 (42) .07 
IDH1 mutated, n (%) 12 (22) 8 (22) 4 (21) >.9 
TET2 mutated, n (%) 9 (16) 4 (11) 5 (26) .2 
NRAS mutated, n (%) 7 (13) 7 (19) 0 (0) .08 
WT1 mutated, n (%) 4 (7.3) 3 (8.3) 1 (5.3) >.9 
PTPN11 mutated, n (%) 4 (7.3) 3 (8.3) 1 (5.3) >.9 
CharacteristicOverall, N = 55Intense, n = 36VEN/HMA, n = 19P value
Sex, n (%)    .6 
Male 27 (49) 19 (53) 8 (42)  
Female 28 (51) 17 (47) 11 (58)  
Race, n (%)    .8 
White 40 (73) 26 (72) 14 (74)  
Black or African American 10 (18) 6 (17) 4 (21)  
Asian/Pacific Islander 5 (9.1) 4 (11) 1 (5.3)  
Age at diagnosis, mean (SD) 67.3 (4.3) 66.1 (3.9) 69.6 (4.2) .005 
ECOG, n (%)    .29 
0-1 43 (81) 31 (86) 14 (74)  
2-3 10 (19) 5 (14) 5 (26)  
CCI, mean (SD) 5.1 (1.8) 4.5 (0.9) 6.2 (2.6) .003 
Treatment era, n (%)     .001 
Treatment era 1 36 (65) 29 (81) 7 (37)  
Treatment era 2 19 (35) 7 (19) 12 (63)  
WBC count at diagnosis, ×103/μL, mean (SD) 65 (78) 74 (82) 47 (67) .12 
Extramedullary disease present, n (%) 18 (33) 12 (33) 6 (32) .9 
Therapy-related, n (%) 7 (13) 4 (11) 3 (16) .7 
Myelodysplastic-related, n (%) 14 (25) 10 (28) 4 (21) .7 
MDS-associated mutation, n (%) 17 (45) 8 (38) 9 (53) .4 
FLT3-TKD mutated, n (%) 19 (35) 12 (33) 7 (37) .8 
DNMT3A mutated, n (%) 16 (29) 8 (22) 8 (42) .12 
IDH2 mutated, n (%) 15 (27) 7 (19) 8 (42) .07 
IDH1 mutated, n (%) 12 (22) 8 (22) 4 (21) >.9 
TET2 mutated, n (%) 9 (16) 4 (11) 5 (26) .2 
NRAS mutated, n (%) 7 (13) 7 (19) 0 (0) .08 
WT1 mutated, n (%) 4 (7.3) 3 (8.3) 1 (5.3) >.9 
PTPN11 mutated, n (%) 4 (7.3) 3 (8.3) 1 (5.3) >.9 

FLT3-TKD, FLT3- tyrosine kinase domain; MDS, myelodysplastic syndrome; SD, standard deviation.

Treatment era 1 are patients with first treatment date before 20 October 2020. Treatment era 2 are patients with first treatment date on or after 20 October 2020.

Myelodysplastic-related refers to patients with AML arising out of prior MDS or MDS/myeloproliferative neoplasm. Because these are favorable risk, by definition they do not have MDS-related cytogenetic changes.

MDS-associated mutations are ASXL1, BCOR, EZH2, RUNX1, SF3B1, SRSF2, STAG2, U2AF1, and ZRSR2.

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